Intracellular malaria parasites export numerous proteins into their host cell, a process essential
for parasite survival and virulence. Many of these proteins are defined by a short amino …

… The distinct, specialized PVMs of Plasmodium and T. gondii correlate to precise adaptations
to their respective intracellular niches (Lingelbach and Joiner, 1998, Cesbron-Delauw et al., …

… Recent work has shed light on endocytic processes in the apicomplexan parasites Plasmodium
falciparum and T. gondii, the causative agents of malaria and toxoplasmosis, respectively…

… Unpaired, two-tailed t test. P values and percent reduction of the mean are indicated. (E)
RSAs after inactivating Kelch13 by KS (rapa) compared with control with Kelch13 3xNLS …

Blood stages of Plasmodium falciparum cause the pathology of malaria; however, the progression
of the parasite through this complex part of the life cycle has never been visualized. In …

Functions have yet to be defined for the majority of genes of Plasmodium falciparum, the
agent responsible for the most serious form of human malaria. Here we report changes in P. …

Current systems to study essential genes in the human malaria parasite Plasmodium falciparum
are often inefficient and time intensive, and they depend on the genetic modification of …

Malaria blood stage parasites export a large number of proteins into their host erythrocyte to
change it from a container of predominantly hemoglobin optimized for the transport of …

For proliferation, the malaria parasite Plasmodium falciparum needs to modify the infected
host cell extensively. To achieve this, the parasite exports proteins containing a Plasmodium …

After invasion of erythrocytes, the human malaria parasitePlasmodium falciparum resides
within a parasitophorous vacuole and develops from morphologically and metabolically …